ĬD 4: 1981's Can't We Fall in Love Again featured Phyllis' highest-charting R&B hit, the title duet with Michael Henderson. This expanded edition includes the Thom Bell-produced "Magic Mona" three tracks on which Phyllis guested with jazz pianist McCoy Tyner and "In A Sentimental Mood" from the Duke Ellington Broadway musical Sophisticated Ladies, in which Phyllis starred and was nominated for a Tony Award. in 1978 this expanded edition features bonus tracks from Sing a Song and a guest appearance with saxophonist Pharoah Sanders, "As You Are," as well as 12" single versions of "Kiss You All Over" and "So Strange." ĬD 3: You Know How to Love Me teamed Phyllis with James Mtume and Reggie Lucas, hitmakers for Stephanie Mills the result was Phyllis' biggest R&B/dance hit with the now-classic title track. The title cut was co-produced by Barry Manilow six tracks were included from Phyllis' second Buddah LP Sing a Songwhich only gained release outside the U.S. ĬD 2: Somewhere in My Lifetime marked Phyllis' addition to the Arista Records roster when Buddah Records was acquired by the Clive Davis-helmed label. Also on this expanded edition, both sides of Phyllis' 1976 single for Desert Moon Records. R&B/Dance charted single, "Loving You, Losing You," co-penned by Philly soul maestro Thom Bell who also co-wrote "Betcha By Golly Wow," which Phyllis recorded with jazz drummer Norman Connors in 1976 along with a duet with Michael Henderson ("We Both Need Each Other"), resulting in her first exposure to a wider audience. Within the brackets, we've added background information and notes to each title.ĬD 1: Phyllis Hyman (Buddah) marked Phyllis' official album debut and includes the U.S. (Watch this space for news of similar SoulMusic boxes coming from Deniece Williams and Atlantic Starr.) This release follows SoulMusic's 2017 two-disc anthology of Hyman's Buddah and Arista recordings.īelow, you'll find Cherry Red's descriptions for each of the nine albums included in Old Friend. Many of those have been reconfigured from past reissues on SoulMusic and other labels. (Hyman's second Buddah LP, Sing a Song, isn't included in its original incarnation but all of its tracks are part of the set.) It's very much in the mold of Cherry Red/SoulMusic's 2020 box for Phyllis' onetime Arista labelmate Dionne Warwick, with expanded editions of numerous albums. The 9-CD box traces Hyman's musical journey by bringing together her studio albums as originally released on the Buddah, Arista, and Philadelphia International labels plus select bonus tracks including collaborations with Barry Manilow, Norman Connors, McCoy Tyner, Pharoah Sanders, Grover Washington Jr., and Michael Henderson. On July 30, Cherry Red's SoulMusic imprint will release Old Friend: The Deluxe Collection 1976-1998. When collaborating with her, Bell channeled all aspects of her sound and persona - from the icy mystique of "Magic Mona" to the vulnerability and longing of "Old Friend." Now, both of those songs - and 111 others - are being collected on the late vocalist's first ever box set. She took on the sound of Nancy Wilson." Indeed, Phyllis built upon Wilson's elegant style with a depth of emotion underneath the singer's cool veneer and towering height, there was a passionate heart and tremendous complexity. "She was a lonely individual," observed Thom, "which in turn enhanced the lonely vibes of a melody - if there was loneliness involved. The songwriter-arranger-conductor-producer would cross paths numerous times over the years with his childhood friend: first via Phyllis' hit recordings of his "Betcha By Golly Wow" and "Loving You - Losing You," and later, his own productions and songs for her. I can still see the pigtails." In a 2016 interview with The Second Disc, Thom Bell shared his earliest memories of the late Phyllis Hyman (1949-1995).
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The way Ludger works his way through conversations, and who he chooses to pair-up with in battle, can build his affinity with other characters, providing a great reason to carefully navigate Xillia 2's extensive exposition while testing out new match-ups during fights. Xillia 2, on the other hand, is far more linear in its campaign's delivery, instead giving Ludger a litany of decisions to make during conversations and combat alike. The biggest turn is the deviation away from Xillia's binary campaign progression, which relied a great deal on whether you were playing primarily as Milla or Jude. For as much as Xillia 2 borrows from Xillia, it takes some smart left turns that ultimately keep the experience new, fresh, and on equal footing with the quality of the original. Her morbidly obese cat companion Rollo, on the other hand, is absolutely amazing. She's essential to the overarching plot, but I always found myself caring about the original cast more, and her subpar voice acting didn't exactly help (nor did Milla's voice actresses' still-disjointed performance, which isn't quite as bad as the original, but still noticeably worse than expert performances from the likes of Alvin's and Rowen's respective actors). Elle, on the other hand, only takes part in the story, and not in combat, which lessened my attachment to her. They’re both admittedly weaker than the main cast from the original game, and while Ludger is interesting, I have no idea why he was designed to be virtually silent. Rounding the group out is Xillia 2's main - and mostly quiet - protagonist, Ludger, and a young, mysterious little girl named Elle. Representing Rieze Maxia are most of the characters you're familiar with from Xillia: Milla, Jude, Leia, Elize, Alvin, Rowen, and others. 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Using this script you can manually reset your The Crafting Dead map and start over with the pack on a fresh new world The Crafting Dead : Origins Instructions Servers Instructions How to reset and change the The Crafting Dead map Map reset script After characterizing eight such mini-PCDH15 versions in vitro and optimizing AAV expression cassettes for inner ear hair cells, we tested their function in vivo in three USH1F mouse models. In this work, we used our structural knowledge of PCDH15 to eliminate 3–5 EC repeats while retaining PCDH15 function, in order to package the coding sequence into a single AAV capsid. PCDH15-CD2 has a ~5.3 kb coding sequence, too large for an AAV capsid. PCDH15 has three alternatively spliced C-termini: CD1, CD2 and CD3, but only the CD2 splice form is necessary for PCDH15 function in hair cells in mice 23, 25. USH1F represents roughly 3–11% of all Usher syndrome cases 36, 37. Additionally, USH1 patients develop prepubertal retinitis pigmentosa (RP), which progresses to nearly complete blindness by the fifth decade 34, 35. Patients with the most severe forms of Usher syndrome, known as type 1 (USH1), experience profound congenital hearing loss and severe balance deficits. It is both genetically and clinically heterogeneous. Usher syndrome (USH) is the most common cause of deaf-blindness worldwide. In humans, mutations in PCDH15 can cause isolated deafness or Usher syndrome type 1 F (USH1F). It also forms transient lateral and kinocilial links, critical for hair bundle development and planar cell polarity 17, 21, 25, and is expressed in retinal photoreceptors and in auditory cortex interneurons 32, 33. Serving as the lower portion of the tip link in mature inner ear hair cells, PCDH15 binds to cadherin 23 (CDH23) 22, 26 to convey force from sound stimuli to the mechanosensory transduction channels 30, 31. This protein is a large, atypical cadherin with 11 link-like extracellular cadherin (EC) repeats and a membrane adjacent domain (MAD12) preceding its single transmembrane domain 22, 29. Over the past decade, we and others have elucidated the molecular structure, function, and interactome of PCDH15 17, 18, 19, 20, 21, 22, 23, 24, 25, 26, 27, 28. However a significant challenge for minigenes is the need to understand the protein structure and binding partners well enough to delete non-essential domains while preserving function, especially when function may include resisting large forces. More recently, mini-otoferlin genes have been used to probe the role of the protein in hair-cell endocytosis in the cochlea 13, 14, mini- CEP290 genes have been evaluated for treating CEP290-associated Leber congenital amaurosis 15, and a “condensed” form of human tuberin (cTuberin) designed for treating tuberous sclerosis complex 16. This strategy has been employed to develop mini- and micro-dystrophins to treat Duchenne muscular dystrophy, based on the observation that large deletions in the dystrophin gene are generally well tolerated 12. Dual and triple AAV approaches are options for circumventing this drawback however they rely on co-transduction of multiple viral particles in the same cell, and on efficient intracellular recombination of the viral DNA or expressed proteins 9, 10, 11.Ī less common approach is to use rational protein design to generate shortened versions of the protein of interest. Elements such as promoters, polyadenylation sequences, and other regulatory elements further limit the size of transgenes that can be packaged. However AAVs remain limited by their capsid packaging capacity, which only allows transport of ~5 kb of genetic material. Capsid design and discovery efforts have dramatically improved AAV transduction rates, particularly in neuronal tissues 1, 2, 3, 4, 5, 6, 7, 8. 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